| （1.郑州大学 体育学院，河南 郑州 450044；2.重庆师范大学 体育学院，重庆 401331） 摘 要：分析运动后收缩压恢复(PSBPR)与单纯性肥胖(超重)的关系，探索PSBPR延迟作为肥胖(超重)患者新的调控标志物的可行性。将169名志愿者按照身体质量指数(BMI)分为肥胖(超重)组(BMI≥24 kg/m2，n=91名)和正常体重组(18 kg/m2≤BMI<24 kg/m2，n=78名)。所有受试者均进行一次递增负荷功率车实验，分别测定运动前安静时、运动中以及运动后恢复期的收缩压，以运动后第3分钟的收缩压与运动后第1分钟收缩压的比值(第3分钟收缩压恢复比，rSBPR)超过1.0作为PSBPR延迟的指标。比较两组rSBPR和PSBPR延迟发生率的差别并分析肥胖指数(体脂百分比、身体质量指数(BMI)、腰围(WC)、腰臀比(WHR)、腰围身高比(WSR))与rSBPR的相关性。结果：肥胖(超重)组rSBPR(男：0.97±0.03；女：0.95±0.05)高于正常体重组(男：0.79±0.02；女：0.80±0.04)(均为P<0.01)；肥胖(超重)组PSBPR延迟发生率(男：16.0%；女：23.4%)显著高于正常体重组(男：2.7%；女：4.3%)(均为P<0.01)；肥胖(超重)的男性和女性发生PSBPR延迟的危险性分别是正常体重者的8.0倍和4.6倍。相关分析显示，肥胖(超重)组男性体脂百分数、BMI与rSBPR相关系数分别为0.381(P<0.01)和0.316(P<0.01)，肥胖(超重)组女性WSR与rSBPR相关系数为0.402(P<0.01)，正常体重组男性体脂百分数、BMI与rSBPR相关系数分别为0.436(P<0.01)和0.310(P<0.01)。结果表明：PSBPR延迟可能是肥胖(超重)患者可调控的心血管危险因素 |
CHEN Lei1，SHI Man-li2
（1.School of Physical Education，Zhengzhou University，Zhengzhou 450044，China；2.School of Physical Education，Chongqing Normal University，Chongqing 401331，China） Abstract: In order to analyze the relations between post-exercising systolic blood pressure recovery (PSBPR) and simple obesity/overweight, and to probe into feasibility to use PSBR delay as a new controllable marker for obe-sity/overweight sufferers, the authors divided 169 volunteers into an obesity (overweight) groups (BMI≥24 kg/m2, n=91) and a normal body weight group (18 kg/m2≦BMI＜24 kg/m2, n=78) based on body mass index (BMI), put all the testees under a load gradually increased power bike experiment, respectively measured systolic blood pres-sure at calm before exercising, during exercising and in the recovery period after exercising, used the ratio of the systolic blood pressure in the 3rd minute after exercising to the systolic blood pressure in the 1st minute after exer-cising (systolic blood pressure recovery ratio in the 3rd minute, rSBPR) exceeding 1.0 as the index of PSBPR delay, compared differences in rSBPR and PSBPR delay occurrence rate between the two groups, analyzed the correlations between obesity indexes (body fat percentage, body mass index, BMI), waist circumference (WC), waist to hip ratio (WHR), waist circumference to stature ratio (WSR)) and rSBPR, and revealed the following findings: the rSBPRs of testees in the obesity (overweight) group (male: 0.97±0.03; female: 0.95±0.05) were higher than those of testees in the normal body weight group (male: 0.79±0.02; female: 0.80±0.04) (P<0.01 for all); the PSBPR delay occurrence rates of testees in the obesity (overweight) group (male: 16.0%; female: 23.4%) were significantly higher than those of testees in the normal weight group (male: 2.7%; female: 4.3%) (P<0.01 for all); the dangers of PSBPR delay oc-curring to obesity (overweight) males and females were respectively 8.0 and 4.6 times those occurring to body weight normal people. Correlativity analysis showed the followings: the coefficient of correlation between WSR and rSBPR of females in the obesity (overweight) group was 0.402 (P<0.01); the coefficients of correlation between BF%, BMI and rSBPR of males in the normal body weight group were respectively 0.436 (P<0.01) and 0.310 (P<0.01). The results indicated that PSBPR delay might be a controllable cardiovascular danger factor for obe-sity/overweight sufferers. |
下载本期全文：点击下载